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Does indomethacin for closure of patent ductus arteriosus affect cerebral function?
Department of Pediatrics, Queen Silvia Children's Hospital, Sahlgrenska University Hospital-Östra.
Department of Pediatrics, Queen Silvia Children's Hospital, Sahlgrenska University Hospital-Östra.
School of Engineering, University of Borås.
Neoventa Medical AB, Göteborg.
Vise andre og tillknytning
2010 (engelsk)Inngår i: Acta Paediatrica, ISSN 0803-5253, E-ISSN 1651-2227, Vol. 99, nr 10, s. 1493-1497Artikkel i tidsskrift (Fagfellevurdert) Published
Abstract [en]

Objective: To study whether indomethacin used in conventional dose for closure of patent ductus arteriosus affects cerebral function measured by Electroencephalograms (EEG) evaluated by quantitative measures. Study design: Seven premature neonates with haemodynamically significant persistent ductus arteriosus were recruited. EEG were recorded before, during and after an intravenous infusion of 0.2 mg/kg indomethacin over 10 min. The EEG was analysed by two methods with different degrees of complexity for the amount of low-activity periods (LAP, "suppressions") as an indicator of affection of cerebral function. Results: Neither of the two methods identified any change in the amount of LAPs in the EEG as compared to before the indomethacin infusion. Conclusion: Indomethacin in conventional dose for closure of patent ductus arteriosus does not affect cerebral function as evaluated by quantitative EEG.

sted, utgiver, år, opplag, sider
2010. Vol. 99, nr 10, s. 1493-1497
Emneord [en]
BIRTH-WEIGHT INFANTS, PRETERM INFANTS, INTRAVENTRICULAR HEMORRHAGE, PREMATURE-INFANTS, HEMODYNAMICS, TRIAL, OXYGENATION, PREVENTION, IBUPROFEN, PROPHYLAXIS
HSV kategori
Identifikatorer
URN: urn:nbn:se:kth:diva-26714DOI: 10.1111/j.1651-2227.2010.01857.xISI: 000281556700016Scopus ID: 2-s2.0-77956357318OAI: oai:DiVA.org:kth-26714DiVA, id: diva2:372670
Merknad

QC 20150727

Tilgjengelig fra: 2010-11-26 Laget: 2010-11-26 Sist oppdatert: 2017-12-12bibliografisk kontrollert

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